Jump to content

42. Oncogenes, protooncogenes, oncoproteins. Growth factor and growth factor receptor oncogenes (RET, KIT, PDGFR). Overexpression of normal growth factor receptors (ERBB1, ERBB2). Organ examples.: Difference between revisions

42. Oncogenes, protooncogenes, oncoproteins. Growth factor and growth factor receptor oncogenes (RET, KIT, PDGFR). Overexpression of normal growth factor receptors (ERBB1, ERBB2). Organ examples. (view source)

Revision as of 22:15, 7 May 2022

8,071 bytes added ,  7 May 2022
Created page with "''I recommend starting with the relevant medical biochemistry topic (27 - 28) before getting into this''. All cancers start with some genetic damage. This genetic damage is always ''nonlethal'', meaning that the damage isn’t big enough to actually kill the cell, however it is definitely big enough to cause some serious damage. The process where cancer develops is called ''carcinogenesis''. Nonlethal genetic damage causes mutations and can be due to many factors. Comm..."
(Created page with "''I recommend starting with the relevant medical biochemistry topic (27 - 28) before getting into this''. All cancers start with some genetic damage. This genetic damage is always ''nonlethal'', meaning that the damage isn’t big enough to actually kill the cell, however it is definitely big enough to cause some serious damage. The process where cancer develops is called ''carcinogenesis''. Nonlethal genetic damage causes mutations and can be due to many factors. Comm...")
(No difference)